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Cardiology update OSCE - KIMS 15 Aug 2026

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 OSCE 1 BY DR. SATYA MURTHY @ APOLLO  X-ray and echo of Anterior displacement of septal tricuspid leaflet - EBSTEINS ANOMALY PR-Prolongation, q in V1Transposition of Great vessels  Oximetry Ruptured sinus or valsalva into RA Pressure tracing PA-RV withdrawal  Valve + infundibular stenosis  * Intact IVS - sharp tip of tracing  With VSD wide tip of tracing  INTRA cardiac ECG look on RA & RV pressures  Ventriculization - EBSTEINS ANAMOLY RA & RV pressure tracings - Tricuspid stenosis CARPENTEER RING STAR EDWARD VALVE ( BALL TYPE - both opening and closing clicks ) PDA BALLON INFLATION ( BALLON OCCLUSION test ) CRISIERS DEVICE MV  SADDLE EMBOLISM  VDD pacemaker  TAPVC - SUPRA CARDIAC PULMONARY AV FISTULA  TOF WITH ABSENT PULMONARY VALVE  Scimitar synd  HCM ALCOHOL SRPTAL ABLATION

Electrocardiographic Diagnosis of Ventricular Tachycardia: A Comprehensive Analysis of Criteria, Algorithms, and Emerging Technologies

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Introduction to Wide Complex Tachycardia Differentiation The accurate interpretation of a wide QRS complex tachycardia (WCT) remains one of the most critical and intellectually demanding exercises in clinical electrocardiography and emergency cardiovascular care. Defined formally as a cardiac rhythm exceeding 100 beats per minute with a QRS duration of 120 milliseconds or greater, WCT primarily encompasses two distinct, life-threatening physiological entities: ventricular tachycardia (VT) and supraventricular tachycardia (SVT) with aberrant intraventricular conduction. The clinical imperative to differentiate these entities with absolute precision stems from the stark differences in their prognostic implications, underlying substrates, and immediate management strategies. Misclassifying VT as SVT with aberrancy can result in the catastrophic administration of atrioventricular (AV) nodal blocking agents, such as verapamil, diltiazem, or beta-blockers, which may precipitate severe hemo...